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Voice of America - English: Biden Picks Senator Kamala Harris as Running Mate

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Presumptive Democratic presidential nominee Joe Biden has chosen California Sen. Kamala Harris as his running mate, making her the first Black woman and the first person of Indian descent to be nominated for a major party’s ticket.

“I have the great honor to announce that I’ve picked @KamalaHarris — a fearless fighter for the little guy, and one of the country’s finest public servants — as my running mate,” Biden tweeted Tuesday afternoon. “Together, with you, we’re going to beat Trump.”

Harris was one of nearly two dozen presidential candidates who challenged Biden for the 2020 Democratic nomination.

After winning enough primaries to secure the nomination, Biden promised to pick a female vice presidential candidate. There was much speculation he would choose a Black woman to run with him.

Former Vice President Joe Biden and U.S. Senator Kamala Harris shake hands before the start of the second night of the second U…
Former Vice President Joe Biden and Senator Kamala Harris shake hands before the start of the second night of the second 2020 presidential Democratic candidates debate in Detroit, July 31, 2019.

Harris, 55, was born in California to a Jamaican-born father and an Indian American mother. She gained prominence after she was elected California’s attorney general in 2010, winning the praise of civil rights activists for refusing to defend the state’s Proposition 8 ballot that banned same-sex marriage.

Her presidential campaign faded quickly when many progressives and liberals questioned her staunch support for law enforcement while attorney general. She was accused of being out of touch over issues of police violence — questions that are bound to be raised again as Americans protest police brutality against Black people and other people of color.

According to some polls, many Black voters hoped Biden would choose the more progressive Massachusetts Sen. Elizabeth Warren as his running mate.

Harris is the third woman to run for vice president on a major party ticket.

Democrat Geraldine Ferraro was Walter Mondale's running mate in 1984 when they lost by a landslide to the Republican ticket of Ronald Reagan and George H.W. Bush.

Former Alaska Gov. Sarah Palin ran with Republican John McCain in 2008 but lost to Democrats Barack Obama and Biden.

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5:30 PM 8/11/2020 - News Review: How Munich Turned Its Coronavirus Outbreak Into a Scientific Study | The New Yorker

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5:30 PM 8/11/2020 - How Munich Turned Its Coronavirus Outbreak Into a Scientific Study | The New Yorker

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                  (3) Michael Novakhov on Twitter: "##FBI is controlled by the #Russian #OrganizedCrime, that is why they were not able to foresee "#Covid19" as the global act of #bioterrorism, and that is why they are not willing and able to deal with it. #InvestigateTheI
                    (3) Michael Novakhov on Twitter: "FBI is controlled by the Russian Organized Crime, that is why they were not able to foresee "Covid-19" as the global act of bioterrorism, and that is why they are not willing and able to deal with it. Investigate the Inve
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                                                      Putin, the Global Firesetter In Chief, turned on his "Firefighting Mode", now he is The Anti-Covid Knight In Shining Armor. Investigate Putin, the New Abwehr German agent, his KGB-s, his GRU-s, his Mafia State, and his Russian Mafia: a bunch of hateful, d
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                                                                                    Scientists debunk a common theory about coronavirus
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                                                                                    CBSNewsOnline's YouTube Videos: Concerns about COVID-19 in children

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                                                                                    Dr. Dyan Hes, a pediatrician in New York City, joins CBSN to discuss the challenges of going back to school amid the coronavirus pandemic, plus a new study that looks at the racial disparities among children hospitalized with COVID-19.

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                                                                                    Scientists debunk a common theory about coronavirus

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                                                                                    Don’t shake hands, don’t high-five, and definitely don’t hug.

                                                                                    We’ve been bombarded with these messages during the pandemic as a way to slow the spread of Covid-19, meaning we may not have hugged our friends or family in months.

                                                                                    This might be really hard for a lot of us, particularly if we live alone. This is because positive physical touch can make us feel good. It boosts levels of hormones and neurotransmitters that promote mental well-being, is involved in bonding, and can help reduce stress.

                                                                                    So how can we cope with a lack of touch?

                                                                                    Touch helps us bond

                                                                                    In humans, the hormone oxytocin is released during hugging, touching, and orgasm. Oxytocin also acts as a neuropeptide, which are small molecules used in brain communication.

                                                                                    It is involved in social recognition and bonding, such as between parents and children. It may also be involved in generosity and the formation of trust between people.

                                                                                    Touch also helps reduce anxiety. When premature babies are held by their mothers, both infants and mothers show a decrease in cortisol, a hormone involved in the stress response.

                                                                                    Touch promotes mental well-being

                                                                                    In adults with advanced cancer, massages, or simple touch can reduce pain and improve mood. Massage therapy has been shown to increase levels of dopamine, a neurotransmitter (one of the body’s chemical messengers) involved in satisfaction, motivation, and pleasure. Dopamine is even released when we anticipate pleasurable activities such as eating and sex.

                                                                                    Disruptions to normal dopamine levels are linked to a range of mental illnesses, including schizophrenia, depression, and addiction.

                                                                                    Serotonin is another neurotransmitter that promotes feelings of well-being and happiness. Positive touch boosts the release of serotonin, which corresponds with reductions in cortisol.

                                                                                    Serotonin is also important for immune system function, and touch has been found to improve our immune system response.

                                                                                    Symptoms of depression and suicidal behavior are associated with disruptions in normal serotonin levels.

                                                                                    But what about a lack of touch?

                                                                                    Due to social distancing measures during the COVID-19 pandemic, we should be vigilant about the possible effects of a lack of physical touch, on mental health.

                                                                                    It is not ethical to experimentally deprive people of touch. Several studies have explored the impacts of naturally occurring reduced physical touch.

                                                                                    For example, living in institutional care and receiving reduced positive touch from caregivers is associated with cognitive and developmental delays in children. These delays can persist for many years after adoption.

                                                                                    Less physical touch has also been linked with a higher likelihood of aggressive behavior. One study observed preschool children in playgrounds with their parents and peers, in both the US and France, and found that parents from the US touched their children less than French parents. It also found the children from the US displayed more aggressive behavior towards their parents and peers, compared to preschoolers in France.

                                                                                    Another study observed adolescents from the US and France interacting with their peers. The American kids showed more aggressive verbal and physical behavior than French adolescents, who engaged in more physical touch, although there may also be other factors that contribute to different levels of aggression in young people from different cultures.

                                                                                    Maintain touch where we can

                                                                                    We can maintain touch with the people we live with even if we are not getting our usual level of physical contact elsewhere. Making time for a hug with family members can even help with promoting a positive mood during the conflict. Hugging is associated with smaller decreases in positive emotions and can lessen the impact of negative emotions in times of conflict.

                                                                                    In children, positive touch is correlated with more self-control, happiness, and pro-social skills, which are behaviors intended to benefit others. People who received more affection in childhood behave more pro-socially in adulthood and also have more secure attachments, meaning they display more positive views of themselves, others, and relationships.

                                                                                    Pets can help

                                                                                    Petting animals can increase levels of oxytocin and decrease cortisol, so you can still get your fill of touch by interacting with your pets. Pets can reduce stress, anxiety, depression, and improve overall health.

                                                                                    In pediatric hospital settings, pet therapy results in improvements in mood. In adults, companion animals can decrease mental distress in people experiencing social exclusion.

                                                                                    What if I live alone?

                                                                                    If you live alone, and you don’t have any pets, don’t despair. There are many ways to promote mental health and well-being even in the absence of a good hug.

                                                                                    The American College of Lifestyle Medicine highlights six areas for us to invest in to promote or improve our mental health: sleep, nutrition, social connectedness, exercise, stress management, and avoiding risky substance use. Stress management techniques that use breathing or relaxation may be a way to nurture your body when touch and hugs aren’t available.

                                                                                    Staying in touch with friends and loved ones can increase oxytocin and reduce stress by providing the social support we all need during physical distancing.

                                                                                    This article was originally published on The Conversation by Michaela Pascoe, Alexandra Parker, Glen Hosking, and Sarah Dash at Victoria University. Read the original article here.

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                                                                                    covid-19 munich cluster and d614g mutation - Google Search

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                                                                                    The D614G mutation in the SARS-CoV-2 spike protein ...

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                                                                                    Jun 11, 2020 - The D614G mutation in the SARS-CoV-2 spike protein reduces S1 ... The coronavirus spike (S) protein mediates receptor binding and fusion of the viral ... also correlated with increased viral loads in COVID-19 patients22, but ...
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                                                                                    Whole genome and phylogenetic analysis of two SARS-CoV-2 ...

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                                                                                    These three genome sequences were located in a cluster with genomes ... two genome sequences from Germany (EPI_ISL_406862 Bavaria/Munich and ... five sequences characterised outside of China, overall eight missense mutations can ... Compared to the viral genome sequence of the COVID-19 patient in Wuhan, the ...
                                                                                    by P Stefanelli - ‎2020 - ‎Cited by 34 - ‎Related articles

                                                                                    Tracking changes in SARS-CoV-2 Spike: evidence that ...

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                                                                                    Jul 28, 2020 - This research is featured on the COVID-19 research community page ... Evaluating the effects of SARS-CoV-2 Spike mutation D614G on transmissibility ... We find that Spike 614G clusters are introduced in the UK later on average ... which caused a small outbreak in Munich around 20 January [1] and was ...

                                                                                    Supplement IV Dendrogram The Undetected_Early European ...

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                                                                                    May 14, 2020 - Investigating the Undetected Early European Covid-19 Outbreak ... 7) The European A2 clade is characterized by the D614G mutation ... Wuhan descendant cluster ... event: The ISPO Sports Business Trade Fair in Munich?

                                                                                    Supplement III Phylogenetics The Undetected Early European ...

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                                                                                    Jun 3, 2020 - Investigating the Undetected Early European Covid-19 ... was not larger because the D614G mutation is somehow better suited to Europeans, but because ... on the Chinese clade, Spain's infections are clustered around its own European clade. ... III b 3) Stage 3: 28 January in the Munich region of Bavaria.

                                                                                    Genetic cluster analysis of SARS-CoV-2 and the identification ...

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                                                                                    Jun 11, 2020 - A newly emerged coronavirus, SARS-CoV-2, caused severe ... Sequence analysis, alignment and mutation identification ... (n = 19, D614G, L752F, F32I, H655Y, V483A, F157L, V615L, K202N, S939F ... EPI_ISL_406862, Germany / Bavaria / Munich, NC, 2020-01-28, 3 / 99%, △ (C241T, C3037T, A23403G).
                                                                                    by X Yang - ‎2020 - ‎Cited by 2 - ‎Related articles

                                                                                    3:22 PM 5/11/2020 - Coronavirus D614G subtype Founder Effect

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                                                                                    May 11, 2020 - Because of the founder effect, showing that a particular mutation increases ... mikenov on Twitter: Covid-19 Germany Bavaria Munich cluster ...

                                                                                    BREAKING! COVID-19 Warning: Study Shows Of Spike ...

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                                                                                    May 1, 2020 - COVID-19 Warning: Disturbing news is emerging as research findings from a ... The study reveals that viruses bearing the mutation Spike D614G are ... Identify Six Specific Symptom Cluster 'Types' Of COVID-19 Infections.
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                                                                                    NEW ORLEANS — A geneticist at LSU Health says a mutated strain of coronavirus now circulating in the United States may be more infectious. Dr. Lucio Miele ...

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                                                                                    Apr 3, 2020 - Nine patients from this 'Munich cluster' subsequently received treatment at München Klinik Schwabing. "At that point time, we really knew very ...

                                                                                    Gene sleuths are tracking the coronavirus outbreak as it ...

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                                                                                    Mar 4, 2020 - By tracking mutations to the virus as it spreads, scientists are creating a family tree in… ... Our most essential coverage of covid-19 is free, including: ... similar to the Munich cluster, says Trevor Bedford, a researcher at the Fred ...
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                                                                                    The Munich Cluster of Covid-19 – GS | SARS-CoV-2 ... - FBI Reform

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                                                                                    The Munich Cluster of Covid-19 – GS · D614G mutation in the SARS-CoV-2 spike protein – GS. Spike protein mutation takes over. A mutation in the spike protein ...

                                                                                    mikenov on Twitter: D614G mutation in the SARS-CoV-2 spike protein

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                                                                                    mikenov on Twitter: D614G mutation in the SARS-CoV-2 spike protein ... from environmental monitoring | Covid-19 The Munich Cluster Mutation: D614G ...

                                                                                    COVID-19 research update: Original research findings ...

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                                                                                    6 days ago - Original findings on the COVID-19 pandemic are being crowded out by ... the study found that a mutation in the virus that causes an amino acid change in its ... additional experimental and clinical data on the D614G variant of the virus. ... Their technique allowed them to cluster countries according to how ...

                                                                                    30/06/20 06:29 | The Munich Cluster of Covid-19 - GS | SARS ...

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                                                                                    Jun 30, 2020 - ... circulating in northern Italy since December 2019: evidence from environmental monitoring | Covid-19 The Munich Cluster Mutation: D614G |.

                                                                                    Why herd immunity to COVID-19 is reached much earlier than ...

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                                                                                    May 10, 2020 - New COVID-19 cases reported in Stockholm County, Sweden, over the 7 days up to the date shown. ... tracing studies I've seen) was prior to the D614G mutation which may ... too and proposes that separate clusters should be simulated separately. ... Martin Hirte | Kinderarztpraxis München – Corona virus.

                                                                                    The Long Road Toward COVID-19 Herd Immunity ... - Frontiers

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                                                                                    Jul 21, 2020 - Although COVID-19 vaccine development is currently accelerated via ... which differ only by a novel synonymous mutation of position D614G in the S ... ReiThera (Rome, Italy), LEUKOCARE (Munich, Germany), and ... from the guinea ring vaccination, open-label, cluster-randomised trial (Ebola Ca Suffit!)

                                                                                    Biorxiv: ​​​​​​​Spike mutation pipeline reveals the ...

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                                                                                    May 5, 2020 - hat tip Michael Coston Spike mutation pipeline reveals the ... If you are sick, call a COVID-19 (coronavirus) hotline, your medical ... The mutation Spike D614G is of urgent concern; it began spreading in ... I think the first known sequence with D641G(spike)=A23403G was in Munich sampled Jan27 together ...

                                                                                    D614G as the one in 1,300 Aminoacids "natural" random ...

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                                                                                    Jul 3, 2020 - The D614G mutation of SARS-CoV-2 spike protein enhances viral ... 30/06/20 06:29 | The Munich Cluster of Covid-19 - GS | SARS-CoV-2 has ...
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                                                                                    How Munich Turned Its Coronavirus Outbreak Into a Scientific Study

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                                                                                    On a bright morning in May, Isabel Klugherz, a medical student at Ludwig-Maximilians University, in Munich, arrived at the geographical center of the city’s Milbertshofen district. Over the previous month, as Munich’s streets emptied under lockdown orders, an exception had been granted for roving medical students in mint-green scrubs and face masks. The university’s department for infectious diseases was screening for the seroprevalence of SARS-CoV-2—antibody testing—by sending police-escorted teams on the winding pattern of a predetermined algorithm, otherwise known as a random walk. Start in the center of an electoral district, continue past a few houses, cross to the other side of the street, select the third house, and knock. In the past month, Klugherz had knocked on nearly a thousand doors, and residents had been overwhelmingly enthusiastic about participating; one man had pronounced it more important than his Sunday celebration of the Holy Mass. That morning, the algorithm led Klugherz into a quiet road of whitewashed modernist buildings. The day before, she had texted a friend who lived nearby to let him know that she would be in the area. Now, unexpectedly, the algorithm led her to his door. She rang the bell. When her friend answered, and found Klugherz standing outside, he was still in his pajamas.

                                                                                    In late March, the government of the state of Bavaria, which includes Munich, was trying to decide how to respond to what appeared to be an accelerating community spread of COVID-19. Michael Hoelscher, the director of the Department of Infectious Diseases and Tropical Medicine at L.M.U., was involved in official debates about whether to impose a citywide lockdown. That, to him, was a foregone conclusion. Considering the disease’s rate of transmission, under the most extreme scenarios, it could take about two and a half years to reach herd immunity in Germany. “Not an option,” Hoelscher said. What was less clear was how the city could effectively track the disease’s spread. Because so many cases of COVID-19 appeared asymptomatic, as Hoelscher had first noted in a paper published in January, diagnostic testing alone would only provide a partial measurement. “So I said, ‘O.K., we need something,’ he told me. The only way to get an accurate measurement of the epidemic, he reasoned, was to implement wide-scale antibody testing. Within six hours, just before the lockdown was announced, on March 21st, he received a million euros from the government.

                                                                                    Hoelscher is tall, with a wave of blond hair that he keeps brushed back cleanly from his face; on the day we met, he was dressed in slim-fitting jeans and a normcore sweater layered over a blue Oxford shirt. Like many infectious-disease researchers of his generation, Hoelscher began his career working on H.I.V. and AIDS. In the early two-thousands, he ran a study on sex workers and bar workers in Tanzania to learn whether it was possible to be infected with H.I.V. more than once. (Deborah Birx, the coördinator of the White House Coronavirus Task Force, was one of his collaborators.) “Take H.I.V.,” Hoelscher said. “You have the disease, you produce an immune response, and that immune response, No. 1, is not able to kill the virus itself. But it can also not protect you from getting a second H.I.V. infection.” The H.I.V. virus, his team discovered, was able to “hide” itself in the immune response, transcribing its genome into the cells. “So that’s an example where the antibody doesn’t help against it,” he said. “In history so far, we only have been able to produce or manufacture a vaccine if the natural immune response would be able to prevent a secondary infection.”

                                                                                    SARS-CoV-2 is obviously different. But it is also exhibiting unusual features. “It can affect multiple organs,” Hoelscher said. “Not only the lower respiratory tract. It can replicate in the upper respiratory tract, it can most likely replicate in different organs.” The course of the disease, the time that it remains in the body, is long, and in some cases, his team found, the immune response does not develop until nearly two months after an infection. “Absolutely surprising or frightening,” Hoelscher said, “is that there might be really some reason to believe that you cannot eliminate it from your body.”

                                                                                    Not far from Hoelscher’s office, L.M.U. had set up a testing tent near the poplar trees of Leopoldstrasse, an imperious boulevard that runs through town. Test subjects who preferred to have their blood drawn outside of their home could come here instead. Outside the tent’s entrance, I met Michael Pritsch, a young infectious-disease doctor who works in Hoelscher’s lab. Pritsch is helping to run the study, but he had come to the tent to get tested himself. In late April, medical students he’d trained had followed the algorithm and ended up at his front door. “My home was my castle,” Pritsch told me. “When I came home after sixteen hours of work, I had my five minutes of free time.” And then they rang his bell. “Even at home, I’m not without this coronavirus thing,” he said. Two students in scrubs and masks welcomed him into the tent and offered him a new mask. He signed a consent form, which he’d helped write, and then pushed up his sleeve as one of the students knotted a rubber tourniquet around his upper arm. Pritsch suggested, wryly, that he would probably check his own data. (“Don’t write that!” he said.)

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